AIDS and Tuberculosis: A Deadly Liaison (Infection Biology by Stefan H. E. Kaufmann, Bruce D. Walker

By Stefan H. E. Kaufmann, Bruce D. Walker

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Extra info for AIDS and Tuberculosis: A Deadly Liaison (Infection Biology Handbook Series)

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Rowland-Jones, S. E. (2001) Clustered mutations in HIV-1 gag are consistently required for escape from HLA-B27-restricted cytotoxic T lymphocyte responses. J. Infect. , 193, 375–386. J. and Walker, B. D. (2006) PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression. Nature, 443, 350–354. S. D. (2007) Upregulation of CTLA-4 by HIV-specific CD4 þ T cells correlates with disease progression and defines a reversible immune dysfunction. Nat. , 8, 1246–1254.

The antigens, HIV-1 Gag, Pol, and Nef, were selected because they are fairly conserved across different HIV-1 clades and commonly recognized during natural infection. In a promising Phase I trial, the study vaccine elicited immune responses in immunocompetent participants, independently of their Ad5 serostatus [75]. The aim of this collaborative study between Merck and the HIV Vaccine Trials Network (STEP study) was to elicit HIV-1-specific cellular immune responses, with the goal of preventing disease progression, but with little expectation that the vaccine would reduce the acquisition of infection.

Meyer, L. and Rouzioux, C. (2000) Natural history of serum HIV-1 RNA levels in 330 patients with a known date of infection. The SEROCO Study Group. AIDS, 14, 123–131. B. C. (1999) Sex differences in longitudinal human immunodeficiency virus type 1 RNA levels among seroconverters. J. Infect. , 180, 666–672. 63 Prins, M. J. (1997) Comparison of progression and nonprogression in injecting drug users and homosexual men with documented dates of HIV-1 seroconversion. European Seroconverter Study and the Tricontinental Seroconverter Study.

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